Coronary collateral development and arterial stiffness in patients with chronic coronary total occlusions

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dc.contributor.authorBaykan, Ahmet Oytun
dc.contributor.authorGur, Mustafa
dc.contributor.authorAcele, Armagan
dc.contributor.authorSeker, Taner
dc.contributor.authorQuisi, Alaa
dc.contributor.authorYildirim, Arafat
dc.contributor.authorCayli, Murat
dc.date.accessioned2024-04-24T17:20:32Z
dc.date.available2024-04-24T17:20:32Z
dc.date.issued2015
dc.departmentDicle Üniversitesien_US
dc.description.abstractObjective. Coronary collateral circulation (CCC) mainly develops through arteriogenesis in response to shear forces. Increased arterial stiffness (AS) causes decreased coronary perfusion, which may reduce shear stress, arteriogenesis, and thus collateral formation. The aim of this study was to assess the relationship between CCC and AS in patients with chronic coronary total occlusion (CTO). Design. We prospectively enrolled 163 patients with CTO. Patients were divided into two groups according to their Rentrop scores: (a) poorly developed (PD) CCC group (Rentrop 0-1) and (b) well-developed (WD) CCC group (Rentrop 2-3). AS measurements were carried out using a Mobil-O-Graph arteriography system. Results. Fasting glucose, creatinine, uric acid, neutrophil count, and neutrophil-to-lymphocyte ratio were found to be higher in patients with PD-CCC. Moreover, patients with PD-CCC had significantly higher augmentation index (AIx) and pulse wave velocity (PWV) compared with WD-CCC group (27.3 +/- 8.9 vs. 18.7 +/- 7.4, p < 0.001 and 11.7 +/- 2.6 vs. 8.5 +/- 1.4, p < 0.001, respectively). In multivariate logistic regression analysis, PWV, AIx, and serum uric acid were found to be independently associated with the development of CCC. Conclusion. AS parameters, such as AIx and PWV, as well as serum uric acid are independently associated with the development of CCC in stable coronary artery disease with CTO.en_US
dc.identifier.doi10.3109/14017431.2015.1062130
dc.identifier.endpage234en_US
dc.identifier.issn1401-7431
dc.identifier.issn1651-2006
dc.identifier.issue4en_US
dc.identifier.pmid26073524
dc.identifier.scopus2-s2.0-84936743415
dc.identifier.scopusqualityQ2
dc.identifier.startpage228en_US
dc.identifier.urihttps://doi.org/10.3109/14017431.2015.1062130
dc.identifier.urihttps://hdl.handle.net/11468/19117
dc.identifier.volume49en_US
dc.identifier.wosWOS:000360175100008
dc.identifier.wosqualityQ4
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoenen_US
dc.publisherTaylor & Francis Ltden_US
dc.relation.ispartofScandinavian Cardiovascular Journal
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/openAccessen_US
dc.subjectCollateral Circulationen_US
dc.subjectArterial Stiffnessen_US
dc.subjectPulse Wave Velocityen_US
dc.subjectAugmentation Indexen_US
dc.subjectUric Aciden_US
dc.titleCoronary collateral development and arterial stiffness in patients with chronic coronary total occlusionsen_US
dc.titleCoronary collateral development and arterial stiffness in patients with chronic coronary total occlusions
dc.typeArticleen_US

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