The effect of passive smoking on the development of respiratory syncytial virus bronchiolitis

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dc.contributor.authorGürkan, F
dc.contributor.authorKiral, A
dc.contributor.authorDagli, E
dc.contributor.authorKarakoç, F
dc.date.accessioned2024-04-24T16:19:08Z
dc.date.available2024-04-24T16:19:08Z
dc.date.issued2000
dc.departmentDicle Üniversitesien_US
dc.descriptionAnnual Congress of the European-Respiratory-Society -- SEP 07-11, 1996 -- STOCKHOLM, SWEDENen_US
dc.description.abstractIn spite of the increasing evidence that passive smoking increases the incidence of respiratory infections and bronchial hyper-responsiveness, the information about whether exposure to sudden heavy smoke enhances the development of acute respiratory infections in children remains inadequate. In this study, to quantitate the level of exposure to environmental tobacco smoke, in 28 children (age ranging 2-18 months) with respiratory syncytial virus (RSV) bronchiolitis and in 30 children (age ranging between 2-15 months) with non-respiratory symptoms, the serum levels of cotinine, the major metabolite of nicotine, were measured at admission to the emergency department. Parents were asked to fill in a questionnaire about the housing conditions and their smoking habits. Serum samples were taken again from the children with RSV bronchiolitis at their second visit at 1 month after discharge from the hospital. The children with RSV bronchiolitis had higher levels of serum cotinine (mean of 10.8 ng/ml) in the acute stage, compared with post-bronchiolitis stage (mean of 7.4 ng/ml). Moreover, patients admitted with non-respiratory symptoms had significantly lower levels of serum cotinine (mean of 3.9 ng/ml) than both phases of patients with RSV bronchiolitis. Children with RSV bronchiolitis were found to have higher levels of cotinine when either the mother or both of the parents smoked, than the children with non-smoker parents. In conclusion, children admitted to the hospital with RSV bronchiolitis were shown to be acutely exposed to more cigarette smoke after 1 month and much more than the children admitted for non-respiratory diseases. These findings may imply that sudden heavy cigarette smoke exposure may predispose to an acute respiratory infection.en_US
dc.description.sponsorshipEuropean Resp Socen_US
dc.identifier.doi10.1023/A:1007658411953
dc.identifier.endpage468en_US
dc.identifier.issn0393-2990
dc.identifier.issn1573-7284
dc.identifier.issue5en_US
dc.identifier.pmid10997834
dc.identifier.scopus2-s2.0-0033865703
dc.identifier.scopusqualityQ1
dc.identifier.startpage465en_US
dc.identifier.urihttps://doi.org/10.1023/A:1007658411953
dc.identifier.urihttps://hdl.handle.net/11468/16412
dc.identifier.volume16en_US
dc.identifier.wosWOS:000089136100009
dc.identifier.wosqualityQ3
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoenen_US
dc.publisherSpringeren_US
dc.relation.ispartofEuropean Journal of Epidemiology
dc.relation.publicationcategoryKonferans Öğesi - Uluslararası - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectBronchiolitisen_US
dc.subjectCotinineen_US
dc.subjectPassive Smokingen_US
dc.subjectRespiratory Syncytial Virusen_US
dc.titleThe effect of passive smoking on the development of respiratory syncytial virus bronchiolitisen_US
dc.titleThe effect of passive smoking on the development of respiratory syncytial virus bronchiolitis
dc.typeConference Objecten_US

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