Carotid Artery Stiffness in Patients With Neurally Mediated Syncope

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dc.contributor.authorElbey, Mehmet Ali
dc.contributor.authorAtilgan, Zuhal
dc.contributor.authorCil, Habib
dc.contributor.authorKaya, Hasan
dc.contributor.authorErtas, Faruk
dc.contributor.authorAydin, Mesut
dc.contributor.authorOzaydogdu, Necdet
dc.date.accessioned2024-04-24T17:33:18Z
dc.date.available2024-04-24T17:33:18Z
dc.date.issued2012
dc.departmentDicle Üniversitesien_US
dc.description.abstractObjectives-Neurally mediated syncope is defined as a transient loss of blood flow to the brain, resulting in vasodilatation, bradycardia, or both. The pathophysiologic mechanisms of neurally mediated syncope are not clear. In this study, we investigated carotid artery elasticity parameters in patients with neurally mediated syncope. Methods-The study was conducted on 41 patients who were examined by the tilt table test. Group 1 constituted of 21 patients who had a positive response to the tilt table test. Clinical and hemodynamic parameters were compared with patients in a negative tilt table test group (group 2). The systolic and diastolic diameters of the carotid arteries, carotid distensibility, carotid strain, carotid stiffness index, and carotid elastic modulus of the left carotid arteries were calculated by a high-resolution ultrasound device. Results-No differences in hemodynamic variables or clinical parameters were detected between the groups. Carotid distensibility (mean +/- SD, 1.6 +/- 0.6 versus 2.2 +/- 0.8 cm(2) x dyne(-1) x 10(-6); P = .044) and strain (6.8% +/- 1.7% versus 8.2% +/- 1.9%; P = .026) were lower in group 1 than in group 2. The carotid elastic modulus (0.88 +/- 0.33 versus 0.74 +/- 0.25 cm(2) x dyne(-1) x 10(-6); P = .002) and stiffness index (6.7 +/- 0.4 versus 6.2 +/- 0.5; P = .038) were higher in group 1. Conclusions-In this study, we concluded that elastic properties of the carotid artery are impaired in patients with neurally mediated syncope. This finding suggests that impaired carotid arterial elasticity may be a factor in the pathophysiologic mechanisms of neurally mediated syncope.en_US
dc.identifier.endpage1435en_US
dc.identifier.issn0278-4297
dc.identifier.issn1550-9613
dc.identifier.issue9en_US
dc.identifier.pmid22922623
dc.identifier.scopus2-s2.0-84866401575
dc.identifier.scopusqualityQ2
dc.identifier.startpage1431en_US
dc.identifier.urihttps://hdl.handle.net/11468/20605
dc.identifier.volume31en_US
dc.identifier.wosWOS:000308451900013
dc.identifier.wosqualityQ2
dc.indekslendigikaynakWeb of Science
dc.indekslendigikaynakScopus
dc.indekslendigikaynakPubMed
dc.language.isoenen_US
dc.publisherAmer Inst Ultrasound Medicineen_US
dc.relation.ispartofJournal of Ultrasound in Medicine
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectCarotid Sonographyen_US
dc.subjectCarotid Artery Stiffnessen_US
dc.subjectNeurally Mediated Syncopeen_US
dc.subjectTilt Table Testen_US
dc.titleCarotid Artery Stiffness in Patients With Neurally Mediated Syncopeen_US
dc.titleCarotid Artery Stiffness in Patients With Neurally Mediated Syncope
dc.typeArticleen_US

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