Etiopathogenesis in fibromyalgia

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dc.contributor.authorGür A.
dc.date.accessioned2024-04-24T18:43:36Z
dc.date.available2024-04-24T18:43:36Z
dc.date.issued2008
dc.departmentDicle Üniversitesien_US
dc.description.abstractFibromyalgia (FM) is a form of non-articular rheumatism characterized by long term (>3 months) and widespread musculoskeletal pain. Research performed in the past decade has shown that patients with FM frequently have dysfunction of the stress response system. Furthermore, emerginq clues propose that such dysfunction may be important in the pathogenesis of the syndrome. The biophysiology of FM, however, has remained elusive and the treatment remains mainly empirical. There are numerous hypotheses about the pathophysiology of chronic widespread pain and FM. Pain, stiffness and depression in FM could be associated with some signs of inflammatory response system activation. Illumination of the pathophysiologic secrets of FM will sign the most effective treatment regims. This article reviews pain mechanisms and new developments in pathopysiology of fibromyalgia.en_US
dc.identifier.endpage11en_US
dc.identifier.issn1302-0234
dc.identifier.issueSUPPL. 1en_US
dc.identifier.scopus2-s2.0-45249101578en_US
dc.identifier.scopusqualityN/Aen_US
dc.identifier.startpage4en_US
dc.identifier.urihttps://hdl.handle.net/11468/24205
dc.identifier.volume54en_US
dc.indekslendigikaynakScopus
dc.language.isotren_US
dc.relation.ispartofTurkiye Fiziksel Tip ve Rehabilitasyon Dergisien_US
dc.relation.publicationcategoryMakale - Uluslararası Hakemli Dergi - Kurum Öğretim Elemanıen_US
dc.rightsinfo:eu-repo/semantics/closedAccessen_US
dc.subjectCentral Mechanismsen_US
dc.subjectEtiopathogenesisen_US
dc.subjectFibromyalgiaen_US
dc.subjectImmun Mediatorsen_US
dc.subjectPeripheral Mechanismsen_US
dc.titleEtiopathogenesis in fibromyalgiaen_US
dc.title.alternativeFibromiyaljide etiyopatogenezen_US
dc.typeReview Articleen_US

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