Yazar "Bircan, Z" seçeneğine göre listele
Listeleniyor 1 - 12 / 12
Sayfa Başına Sonuç
Sıralama seçenekleri
Öğe Can intravenous cyclophosphamide be used for steroid-dependent nephrotic syndrome?(Springer Verlag, 1997) Bircan, Z; Soran, M; Kervancioglu, M; Gonlusen, G; Tuncer, I[Abstract Not Available]Öğe Childhood idiopathic nephrotic syndrome in Turkey(Blackwell Publishing Asia, 2002) Bircan, Z; Yilmaz, AY; Katar, S; Vitrinel, A; Yildirim, MBackground : It has been reported that there are racial and regional differences in peak incidence age, histopathological features and response to steroid therapy in childhood idiopathic nephrotic syndrome. Methods : One hundred and thirty-eight patients with a diagnosis of idiopathic nephrotic syndrome, followed up in 1994-2000, were assessed retrospectively. The aim of this study was to assess the patients response pattern to steroid therapy, to determine whether the duration of the initial steroid therapy alters the steroid response pattern of the disease and to assess renal biopsy results. Results : One hundred and fourteen patients who initially received only steroid therapy and were followed up regularly were classified according to response pattern. Of the 114 patients, 30 children had an initial response, 25 children had infrequent relapse, 19 had frequent relapse, 25 had steroid dependence and 15 children had steroid resistance. The 99 patients with steroid responsive nephrotic syndrome were divided into two groups with respect to duration of the initial steroid therapy. There was no statistically significant difference between standard and short therapy groups with respect to the steroid response patterns. Percutaneous renal biopsy was performed in 43 of the 138 patients. Mesengioproliferative glomerulonephritis was the most common histopathological lesion, followed by membranoproliferative glomerulonephritis. The proportions of membranous glomerulonephritis, focal segmental glomerulosclerosis and minimal change nephrotic syndrome were low in our group. Conclusions : Our study group is similar to one reported from Saudi Arabia with respect to the steroid response pattern and to Saudi Arabian and Nigerian reports with respect to the histopathology. Although it has been reported that short initial steroid therapy was followed by a higher rate of relapses, there was no statistically significant difference between standard and short therapy groups with respect to the relapse rate in our study group.Öğe Chronic renal failure in south eastern anatolia(Springer Verlag, 1996) Bircan, Z; Bircan, ML; Kervancioglu, M; Kilinc, M; Inci, D[Abstract Not Available]Öğe Does albumin and furosemide therapy affect plasma volume in nephrotic children?(Springer, 2001) Bircan, Z; Kervancioglu, M; Katar, S; Vitrinel, AAlbumin infusions transiently increase plasma volume (PV) and oncotic pressure, and may restore diuretic responsiveness in nephrotic edema. To determine if albumin and furosemide therapy have an effect on PV in nephrotic children, 14 severely edematous children with minimal change nephrotic syndrome were evaluated with standard clinical parameters (heart rate, blood pressure, body weight, pretibial edema, abdominal circumference) and echocardiography [inferior vena cava index (IVCI), inferior vena cava collapsibility index (IVCCI)] before, 1 h and 24 h after albumin (20%, 0.5 g/kg, 1 h) and furosemide (2 mg/kg, IV) therapy. An increase in IVCI (P <0.05), decrease in IVCCI (P<0.05), edema (P0.005), and hematocrit (P <0.005) were statistically significant 1 h after albumin and furosemide therapy, with a transient effect 24 h later. Body weight (P <0.005), abdominal circumference (P <0.05), and edema (P <0.005) decreased significantly at 24 h. It is concluded that albumin and furosemide therapy increases PV transiently in nephrotic edema, returning to baseline values at 24 h with a decrease in body weight, abdominal circumference, and edema.Öğe The effect of alternate-day low-dose prednisolone on bone age in children with steroid-dependent nephrotic syndrome(Springer Verlag, 1996) Bircan, Z; Soran, M; Yildirim, I; Dogan, M; Sahin, A; Bilici, A; Danaci, M[Abstract Not Available]Öğe Growth hormone stimulation tests in chronic renal failure with metabolic acidosis(Blackwell Publishing Asia, 1998) Bircan, Z; Kervancioglu, M; Soran, M; Yildirim, IMetabolic acidosis is one of the possible causes of growth retardation in chronic renal failure (CRF). Data about the effect of metabolic acidosis on growth hormone (GPI) secretion in CRF are limited. A study was carried out on eight CRF patients, hospitalized because of severe metabolic acidosis, and eight age-matched prepubertal healthy short children. Growth hormone stimulation tests were done with L-dopa and clonidine before or during acidosis therapy and after the correction of metabolic acidosis. The levels of GH were measured by radio-immunoassay. The mean of the peaks of both tests were used for each patient and statistical significance was tested by Mann-Whitney U and Wilcoxon tests. No difference was found between the GH peaks of the two groups during acidosis and after the correction of acidosis (Wilcoxon test, P > 0.05). In view of this data it was concluded that metabolic acidosis has little effect on GH stimulation tests.Öğe Is there a relationship between serum essential amino acids and insulin-like growth factor-1 levels in children with chronic renal disease?(Oxford Univ Press, 1998) Buyan, N; Hasanoglu, A; Hasanoglu, E; Biberoglu, G; Cinaz, P; Bircan, Z[Abstract Not Available]Öğe A new treatment for clot retention: Intravesical streptokinase instillation(Williams & Wilkins, 1996) Korkmaz, K; Sahin, H; Islim, F; Bircan, Z; Inci, I[Abstract Not Available]Öğe Serum levels of carnitine, apolipoprotein A I, and apolipoprotein B in children with nephrotic proteinuria(Springer Verlag, 1996) Bircan, Z; Kaplan, A; Soker, M; Batun, S; Sahin, A; Soran, M[Abstract Not Available]Öğe Sialic acid in childhood renal diseases: Correlation with clinical and laboratory indices(Blackwell Science, 1998) Bircan, Z; Batun, S; Kervancioglu, M; Soran, M; Kaplan, A; Onur, H; Demir, FThere are many kinds of glycoproteins that have sialic acid residues and it has been reported that these are elevated in some renal diseases and their significance in the pathogenesis of several renal diseases has been investigated. In the present study the serum and urine levels of sialic acid were measured in healthy controls and in children with either poststreptococcal acute glomerulonephritis (PSAGN) or minimal change nephrotic syndrome (MCNS) to test if there is any correlation with clinical and laboratory indices. In PSAGN and MCNS patients the serum and urine sialic acid concentrations at onset and relapse were significantly different from healthy controls (Mann-Whitney U-test P < 0.005). There was not a significant correlation between the clinical severity, serum creatinine and complement C-3 levels and serum sialic acid concentrations in PSAGN patients. Also there was not a significant correlation between edema, serum albumin, IgG, transferrin, alpha-1-antitrypsin and serum sialic acid concentrations in MCNS patients. Although high serum and urine sialic acid levels were found in both PSAGN and MCNS patients, it does not have any clinical significance nor is it important as a diagnostic or prognostic marker.Öğe Two cases of nephrotic syndrome and tertian malaria in south-eastern Anatolia(Springer Verlag, 1997) Bircan, Z; Kervancioglu, M; Soran, M; Gonlusen, G; Tuncer, ITertian malaria is endemic in south-eastern Anatolia. As in Europe and America, in south-eastern Anatolia, an etiological agent is seldom identified in nephrotic syndrome, Two patients with Plasmodium vivax and nephrotic syndrome are described here. The possible relationship between Plasmodium vivax and nephrotic syndrome should be explored in children in endemic malarial regions.Öğe Urinary system stone disease is endemic in Southeastern Anatolia(Scandinavian University Press, 1997) Sahin, H; Bircan, Z[Abstract Not Available]
